The advance within the treatment of hypopsia caused by Retinal pigment degeneration, degeneration and atrophy of the cranial nerve.
Retinal abnormality
The tissue layer may be a layer of skinny hyaline tissue all of the visual info is transferred to the central system by the nerve cells (cones and rods) within the tissue layer in alternative words, it's extension a part of the central system.
The causes of pathological changes: the foremost common cause is ischaemic retinopathy, eruption visual loss in single eye ought to be extremely suspected of anaemia retinae. detachment of the retina, degeneration, eye disease and vitreous hemorrhage square measure causes of retinopathy. the subsequent can chiefly justify retinosis.
Retinosis
Retinosis is one among the most causes of chronic diminished visual modality, normally it includes membrane degeneration and degeneration. There square measure many sorts that square measure most likely associated with alternative system unwellness.
Macular degeneration is that the main reason for visual loss in old folks, step by step there's a decrease of central visual modality, that affects reading, the patient retains the sight and may still walk.
In young and old folks, the foremost common unwellness may be a reasonably hereditary diseases within the external lightweight layer and also the adjacent pigment epithelial tissue, named inflammation pigmentosa. Clinically the contraction of the field of vision, visual modality relative preservation (tube sort field) visual disorder (visual distortion), poor adaptation to the dark and visual disorder square measure characteristics of this unwellness.
Teen-agers retinal degeneration a sort of staged degeneration unwellness principally seen in teen-agers that main pathological modification is massive areas of central retinal lesions it may result in progressive spastic paralysis or motor ataxia.
In infants and young youngsters, some Lysosomal diseases referred to as degeneration. this is often characterised by supermolecule that can't be degraded and carbohydrate gathering within the brain, cranial nerve, macular and tissue layer.
Retinosis is additionally will seen in oat-cell malignant neoplastic disease patients, those patients bodily fluid have antibodies against retinal neural structure cells.
These square measure a sort of progressive vision loss, visual disorder contraction of field of vision, structure pigmentation hereditary, chromosomal mutation and immune injury of disease. ordinarily seen in heredopathia, modes of inheritance square measure chromosome dominant, recessive or sex chromosome recessive inheritance, most involve each eyes, one eye being concerned and Quadrant square measure rare. The unwellness progresses slowly, with AN unfavourable prognosis. The younger the age of onset, the additional serious, the sooner visual impairment happens. The older the age of onset, the later visual impairment happens.
Pathological change:
The main modification is degeneration within the stratum neuroepitheliale retinae, particularly within the rod cells, than the tissue layer step by step atrophies from the within to the surface, with gliosis.
Degeneration and dysplasia happens within the pigment vegetative cell will realize cases of coloration or accumulation moving to the entorretina. Glassy degeneration and thickening within the retinal tube wall, will even entirely enclose the lumen. totally different degrees of hardening within the choroidal vessels, leads to the blood capillary utterly or part disappearing. The cranial nerve might complete atrophy, gliosis in discus opticus that becomes a membrane, connects with the gliosis within the tissue layer.
Clinical manifestation:
Treatment:
Traditional treatment can't stop degeneration and death in retinal nerve cells and pigment cells. There has been no effective treatment before. In recent years, through quite 10 years treatment expertise, Wu stem cells center (WSCMC) found that stem cells will have an honest treatment outcome in Retinosis. vegetative cell treatment will effectively abate or stop the unwellness progressing. The molecular chaperones from stem cells will clear an area of abnormal accumulation supermolecule, will abate or stop the unwellness progress. And immunoregulation from stem cells will resist the protein that causes injury within the tissue layer and cranial nerve, the neuron that is differentiated from nerve stem cells will repair a part of the injury and also the vitium, then will increase the attention sight and expand the field of vision of the patient.
Retinal abnormality
The tissue layer may be a layer of skinny hyaline tissue all of the visual info is transferred to the central system by the nerve cells (cones and rods) within the tissue layer in alternative words, it's extension a part of the central system.
The causes of pathological changes: the foremost common cause is ischaemic retinopathy, eruption visual loss in single eye ought to be extremely suspected of anaemia retinae. detachment of the retina, degeneration, eye disease and vitreous hemorrhage square measure causes of retinopathy. the subsequent can chiefly justify retinosis.
Retinosis
Retinosis is one among the most causes of chronic diminished visual modality, normally it includes membrane degeneration and degeneration. There square measure many sorts that square measure most likely associated with alternative system unwellness.
Macular degeneration is that the main reason for visual loss in old folks, step by step there's a decrease of central visual modality, that affects reading, the patient retains the sight and may still walk.
In young and old folks, the foremost common unwellness may be a reasonably hereditary diseases within the external lightweight layer and also the adjacent pigment epithelial tissue, named inflammation pigmentosa. Clinically the contraction of the field of vision, visual modality relative preservation (tube sort field) visual disorder (visual distortion), poor adaptation to the dark and visual disorder square measure characteristics of this unwellness.
Teen-agers retinal degeneration a sort of staged degeneration unwellness principally seen in teen-agers that main pathological modification is massive areas of central retinal lesions it may result in progressive spastic paralysis or motor ataxia.
In infants and young youngsters, some Lysosomal diseases referred to as degeneration. this is often characterised by supermolecule that can't be degraded and carbohydrate gathering within the brain, cranial nerve, macular and tissue layer.
Retinosis is additionally will seen in oat-cell malignant neoplastic disease patients, those patients bodily fluid have antibodies against retinal neural structure cells.
These square measure a sort of progressive vision loss, visual disorder contraction of field of vision, structure pigmentation hereditary, chromosomal mutation and immune injury of disease. ordinarily seen in heredopathia, modes of inheritance square measure chromosome dominant, recessive or sex chromosome recessive inheritance, most involve each eyes, one eye being concerned and Quadrant square measure rare. The unwellness progresses slowly, with AN unfavourable prognosis. The younger the age of onset, the additional serious, the sooner visual impairment happens. The older the age of onset, the later visual impairment happens.
Pathological change:
The main modification is degeneration within the stratum neuroepitheliale retinae, particularly within the rod cells, than the tissue layer step by step atrophies from the within to the surface, with gliosis.
Degeneration and dysplasia happens within the pigment vegetative cell will realize cases of coloration or accumulation moving to the entorretina. Glassy degeneration and thickening within the retinal tube wall, will even entirely enclose the lumen. totally different degrees of hardening within the choroidal vessels, leads to the blood capillary utterly or part disappearing. The cranial nerve might complete atrophy, gliosis in discus opticus that becomes a membrane, connects with the gliosis within the tissue layer.
Clinical manifestation:
1. Visual disorder and progressive visual loss.
2. Field of vision progressive concentrical contraction to cannular field of vision.
3. Optic fundus: pale, sallow, retinal tube slender, structure grey cyan contamination, embryonic cell pigmentation, obvious in ambitus, atrophy of the tissue layer, will see sclerotic chorioid.
4. Visible radiation structure angiography: transmitted visible radiation in early stage, blood vessel filling slowly, region might have leak as a result of cystoid macular lump.
5. ERG: destroyed sort.
Treatment:
Traditional treatment can't stop degeneration and death in retinal nerve cells and pigment cells. There has been no effective treatment before. In recent years, through quite 10 years treatment expertise, Wu stem cells center (WSCMC) found that stem cells will have an honest treatment outcome in Retinosis. vegetative cell treatment will effectively abate or stop the unwellness progressing. The molecular chaperones from stem cells will clear an area of abnormal accumulation supermolecule, will abate or stop the unwellness progress. And immunoregulation from stem cells will resist the protein that causes injury within the tissue layer and cranial nerve, the neuron that is differentiated from nerve stem cells will repair a part of the injury and also the vitium, then will increase the attention sight and expand the field of vision of the patient.
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